We did a series of experiments, including wound-disruption studies. We targeted the endothelial cells and we got these from our patients intraoperatively. What we found was that there was a series of factors that seemed to drive tumor angiogenesis, which are missing from normal angiogenesis. We have been in the process of trying to come up with a therapeutic targeting strategy for that specific factor. We know that these are relevant, and we are really excited at the possibility of coming up with a tumor angiogenesis drug that won't affect wound healing.